Why do we need a cure for cancer?

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So you are using an antiparasite medicine Ivermectin to destroy the biome of the solid state cancers?

Ivermectin
When taken for onchocerciasis (river blindness), the rapid death of parasites can trigger a "Mazzotti reaction," resulting in fever, swollen lymph nodes, joint pain, and eye irritation.
>Overdoses or misuse can lead to coma or death

_________

There is a cancer solution which doesn't run these risks

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--> https://pmc.ncbi.nlm.nih.gov/articles/PMC7505114/

from OP link image

>>539522553
Green tea. Shuts down the feeding of cancer cells.

>>539523264
akt/mTOR
Wnt / β-catenin
MAPK
____________
β-catenin
Lycopene: Blocks the movement of β-catenin into the cell nucleus, which stops survival genes like c-myc from turning on in stomach cancer cells.

Astaxanthin: Lowers inflammation and stops harmful Wnt/β-catenin signaling activity linked to tumor progression.beta-

Carotene: Helps regulate and reduce cancer stem-like cell properties in colon tissue models

>>539523359
EGCG (Epigallocatechin gallate) from Green Tea

Epigallocatechin gallate (EGCG), a primary green tea catechin, acts as a direct, ATP-competitive inhibitor of the mammalian target of rapamycin (mTOR) and PI3K pathways. It suppresses cell growth, regulates autophagy, and influences various disease models

>>539523359
>akt/

Epigallocatechin gallate (EGCG)—the main catechin in green tea—modulates the AKT signaling pathway (Protein Kinase B), which controls cell growth, survival, and metabolism.

EGCG and AKT Interactions

Pathway Regulation: EGCG targets the PI3K/AKT/mTOR signaling axis, frequently suppressing phosphorylated AKT (p-Akt) to help inhibit abnormal cell proliferation in research models.Cell Survival: It alters AKT-mediated signaling to induce apoptosis (programmed cell death) in targeted or damaged cells.

>>539523359

MAPK Signaling Modulation

EGCG suppresses activation of MAPK pathways (including ERK1/2, JNK, and p38) to exert anti-inflammatory effects.

It inhibits pro-inflammatory gene expression and oxidative stress damage through these kinase interactions.

good thread
fuck big pharma

In cell biology, mTOR (mechanistic target of rapamycin) is widely considered a master regulator pathway—often called the cell’s central metabolic switchboard.

While the MAPK pathway acts primarily as a high-volume communication pipeline (relaying external signals like growth factors and stress into the cell), mTOR is the system that decides whether the cell actually has the fuel and resources to respond.
What Makes mTOR a Master Pathway?

>If MAPK answers the question "Is there an external signal to grow or react?", mTOR answers the question "Do we have the budget and energy to do it?"

mTOR operates as a central node that integrates four key inputs before making major cellular decisions:

1. Nutrient Availability: Scans for amino acids (especially leucine) and glucose.

2. Energy Status: Monitors ATP levels (interacting closely with AMPK, the cell's fuel gauge).

3. Growth Signals: Receives input from insulin, growth factors, and upstream cascades (like PI3K/Akt and MAPK itself).

4. Stress Levels: Detects hypoxia (low oxygen), DNA damage, and osmotic stress.

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The Two Engine Complexes: mTORC1 vs. mTORC2

mTOR doesn't work alone—it forms two distinct multi-protein complexes that drive different cellular programs:

How Natural Polyphenols Suppress NF-κB

Natural polyphenols—such as EGCG (green tea), Curcumin (turmeric), Resveratrol (grapes), and Quercetin (apples/onions)—act as potent multi-target inhibitors of the NF-κB pathway. Rather than hitting just a single target, they shut down the alarm system at multiple checkpoints:
1. Inhibiting the Master Switch (IKK Kinase)

Polyphenols directly suppress the activity of the IKK complex. By preventing IKK from phosphorylating IκB, the "brake" stays attached to NF-κB, keeping it trapped safely outside the nucleus in its inactive form.
2. Blocking Nuclear Translocation

Even if some NF-κB breaks free, polyphenols can physically impede its translocation across the nuclear membrane, preventing it from accessing the cell's genetic machinery.
3. Neutralizing Upstream Triggers (ROS & Cytokines)

Polyphenols are powerful antioxidants. By scavenging Reactive Oxygen Species (ROS) and downregulating upstream cytokine receptors, they remove the initial stress signals that trigger the pathway in the first place.
4. Direct DNA Binding Interference

Some polyphenols penetrate the nucleus and alter the DNA-binding domain of NF-κB, preventing it from properly attaching to promoter regions on genes even if it reaches the nucleus.

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Primary NF-κB Suppression Mechanism

Polyphenols

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>>539522553

I know plenty of people who've tried all the things you listed and more and have died from cancer. I also know people who've tried completely crazy shit and cured their cancer because of it. I know one guy who survived off nothing but vegetable juice for a year and cured his cancer, literally he'd just juice like 20lbs of vegetables a day and drink it. Nothing else, just that and within a year he was cured.

Cancer isn't one disease. It's a collection of diseases with wildly different causes. There will never be a single cure for it because of this one fact.

>>539527422

Big problem with all of the substances listed in this image is they have incredibly poor bioavailability and the only tests that showed they were effective require the sort of doses that simply aren't possible to achieve.

Curcumin for example. Tumeric is around 3-5% curcumin, and your body can absorb about 0.1-2% of all the curcumin you eat, even with black pepper.
The doses they tested that were effective against cancer would require you to have 10-15 grams of curcumin in your blood stream. To realistically achieve this in the most ideal situation, you'd need to eat 15kg of tumeric a day (that much tumeric would probably poison you, if not it is also over 50,000 calories a day).

This is why you cannot take this shit seriously. No one can eat 15kg of tumeric a day.

>>539531887
>The doses they tested that were effective against cancer would require you to have 10-15 grams of curcumin in your blood stream.

Could you show me this study with the dosage please

>>539531887
I had a study on THC (tetra-hydra-curcumin) I'm trying to find it at the moment.

If you could post the study you are referring to that would be brilliant.

>>539522553
maybe stop eating food from any company with jewish investors. all that product is designed to create diseases and cancer as a form of supply chain management.

>>539531887
No such study exists with those dosages?

>>539522553
>memeflag flooding the thread
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>>539534457

Except they do. People like you are the reason most of these studies are so misunderstood. I specifically said these studies would be like having 10-15 grams of curcumin in your blood stream. I never said they used 10-15 grams of curcumin in the study.

Most of the studies of curcumin are in vitro. They involve giving something like 1mg of curcumin to 100 thousand cancer cells (which is so tiny you couldn't see it). When you scale this up to the human body, you need absolutely collosal doses.
A cancerous tumor about 1cm/3 would have around 1 billion cells. This is 10,000x more than 100,000 cancer cells they might test in a lab. This would mean to achieve a similar effect, instead of 1mg of curcumin, you'd need 10,000mg, which is 10 grams. In your blood stream, all flowing to the tumor.

This is why most studies done in a lab cannot be taken as evidence something works. In almost all cases it's impossible to achieve similar doses in the human body. Either it's just physically impossible, or so dangerous it'd do way more harm than good.

>>539535989
May I see the study you are referring to please