Eternal youth edition. MY CURRENT PROTOCOLDrugs:>Exemestane 6.25mg>Aspirin 300-500mg>Telmisartan 20mg>Montelukast 10mg>Finasteride 1.25mg>Bromocriptine 2.5mg>Ondansetron 4mg>Doxycycline 50mg>Tretinoin 0.1%Base:>Magnesium citrate 1g>Calcium ascorbate 2g>Vitamin E 400mg>Vitamin D 10,000IU>NAC 1g>Vitamin K3 7.5mg>MSM 4g>L-theanine 300mg>Iodine 200mcg
>>77247052>no pitavastatin>no empagliflozinngmi
>>77247052Who cares you all look like skinnyfat indians
>>77247054>pitavastatinDetrimental for muscular hypertrophy and strength. I know we had this debate before, but i can provide new studies to discuss if you want. >empagliflozinNegligible benefits if you're using low-carb diet or perform high-effort physical activity around high-carb meals. I think drugs of gliptin class provide more benefits regarding glucose metabolism by directly upregulating hormones involved in it. >>77247055>you all look like skinnyfat indiansHere's what i look like - 195cm/95kg and 100% aryan.
>>77247069>Detrimental for muscular hypertrophy and strengthWrong and I can gladly cite the MR study again.>but i can provide new studies to discuss if you want.Moreover, with high cholesterol it doesn't matter how many other things you take, you'll still die early.I bet it's another observational trial or worse.>Negligible benefits if you're using low-carb dietA keto diet kills you while empagliflozin protects your kidneys and heart.
>>77247069>Looks like a skinnyfat indian Guessed correctly
>>77247069My friend, you're ugly. Consider plastic surgery.
>>77247120>you're ugly.I'm majestic.
>>77247135Chin bone are good, the rest really need to change.I'd start with your posture, it doesn't take surgery.
>>77247075>I can gladly cite the MR study again.This study haven't showed statins are beneficial or at least neutral for muscles. >I bet it's another observational trial or worse.No. >Simvastatin impairs exercise training adaptationshttps://sci-hub.ru/https://pubmed.ncbi.nlm.nih.gov/23583255/Our findings suggest that simvastatin may mitigate improvements in fitness in response to exercise training by impairing increases in skeletal muscle mitochondrial content and function. In support of these data, physiologic doses of simvastatin disrupt mitochondrial respiration, increase oxidative stress, and activate mitochondrial apoptotic pathways in isolated skeletal muscle fibers (27). Similar observations have been reported in studies of muscle fibers taken from patients using statins (28), and high dose simvastatin (80 mg per day) has been shown to decrease skeletal muscle mitochondrial content in the absence of exercise (29,30). Statins have also been shown to reduce skeletal muscle force production (31), running capacity (17,18) and voluntary running volume (31) in rodents.>empagliflozin protects your kidneys and heart.All it does is lowering blood glucose levels. It's useless if you're not overconsuming carbs or have high level of physical activity when your muscles act as glucose sink, and can lead to hypoglycemia.
>>77247135Negative canthal tiltChud lines nasolabial foldsRecessed jawlinePudgy brow ridgePhiltrum you can play tic tac toe onOverall round potato head faceVerdict : sub5 doomed to a loveless sexless life
>>77247052"Biohacking" is even more cringe than nootropics
>>77247244So true, nattykek bro.
>>77247052The great thing about this retarded shit is that, usually, the problem takes care of itself. You just have to hope these faggots off themselves with their schizo shit before causing too much harm by convincing others to do the same.If you want to spot someone without any degree in any field of biology, or even any schooling in a single course on biology at the college level, that retard faggot list is pretty much it.
>>77247350The amount of ressentiment this threads evoke in lesser men really makes me think biohaking is the new steroids.
>>77247069oh brother
>>77247055
>>77247411True, biohacking is aryan culture.
>>77247370>resentmentFaggot, I don't envy anyone anything. It's literally my fucking job to use endocrinology and neuroscience research to shape policies. All I fucking care about is the application of biological principles to how we live our day-to-day lives in modern society, for the betterment of human functioning. My life revolves around giving people that ability.You're a dangerous schizo.
>>77247135plz fuck me daddy
>>77247435>I don't envy anyone anything.You envy me, like a lesser men should. I humiliated you several times on this board, first time discussing calpain and then discussing aspirin-triggered lipoxins. That's why you're not even trying to debate me anymore and just emotionate.>You're a dangerous schizo.I'm a great alchemist, dangerous indeed.
>>77247069how can you look like a pdf and his victim in the same time???
>>77247411>>77247423Says the slavic potatohead straight from the steppes
Taking an aromatase inhibitor with no test is some fucking retarded shit bro lol. Low estrogen symptoms are often indistinguishable from high estrogen symptoms. You don't want this shit.
>>77247052>>77247069>>77247135
>>77247650Cope.
>>77247530You're correct, look at his physique
>>77247530Exemestane has been the best addition to my stack. Your opinion is skewed by reading reports from users of exogenous androgens and breast cancer patients, as well as use of non-steroidal aromatase inhibitors. In healthy men not using exogenous androgens it acts very differently - exemestane raises total testosterone by 50%, free testosterone by more than 100% and lowers estrogen only by 40%. Here's a good explanation of the mechanism:"AIs absolutely act differently in men who are not HPTA suppressed, due to the actions of an intact negative feedback loop.In men not using exogenous androgens it goes like this...take an AI -> estradiol goes down -> pituitary gland recognizes that estradiol is decreasing -> men only have one way to create more estradiol (aromatization) -> pituitary gland increases gonadotropin production in order to create more testosterone -> this testosterone is aromatized into estradiol.That’s the negative feedback loop at work.When there is an interruption in this loop (gonadotropin production ceases when we use testosterone), that’s when it becomes very easy to drive estradiol too low. Since we are shut down, the pituitary gland is unable to ramp up gonadotropin production in its attempt to create more estradiol via aromatization.There are numerous studies of men on AI monotherapy in which they used very large doses, but did not achieve anywhere near the suppression of estradiol that we would see in somebody using testosterone as well. We’re talking 1-2mg of Arimidex per day, 2.5mg of Letrozole several times per week, etc. What occurred was a significant increase in testosterone levels, and a moderate decrease in estradiol.Here are some relevant studies:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3143915/https://academic.oup.com/jcem/article/88/12/5951/2661508https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2795655/http://press.endocrine.org/doi/10.1210/jcem.85.7.6676https://www.ncbi.nlm.nih.gov/pubmed/11397902 "
>>77247848Too many words to say you're dyel and look like shit
>>77247864>you're dyelI literally don't even lift. My body was built by intense physical labor that i get paid for. >look like shitI mog you.
>>77247530>>77247848>Relevance of anti-inflammatory and antioxidant activities of exemestane and synergism with sulforaphane for disease preventionhttps://pmc.ncbi.nlm.nih.gov/articles/PMC3839725/In conclusion, in addition to its potent mechanism-dependent inhibition of estrogen biosynthesis, exemestane has chemoprotective properties that have hitherto not been explicitly recognized. Exemestane should therefore be considered for use also in chemoprotection against mammary tumors that are not estrogen receptor-positive, and against a wide variety of nonmammary tumors (and possibly other chronic diseases) that are not estrogen-dependent but have oxidative stress, inflammation, and electrophile-damaging etiologies. The additional finding that exemestane shows powerful synergism with another widely consumed Nrf2-activator, sulforaphane, and a number of other phytochemicals, increases the attractiveness of this strategy. Our findings favor the view that use of exemestane with its broad range of actions, and its potential synergism with sulforaphane and other phytochemicals, could be a valuable chemoprotective strategy for reducing the risk of many malignancies and possibly other chronic diseases.
>>77247899>Aromatase inhibitors regenerate the thymus in aging male ratshttps://www.sciencedirect.com/science/article/pii/0192056192901152"...The thymus can be regenerated in aging rats by surgical or chemical castration and regeneration is inhibited by testosterone, which may exert this effect, at least in part, through its conversion to estradiol. An attempt has been made to regenerate the thymus in intact aging rats using inhibitors of the aromatase system, in the hope that this maneuver could lead to the use of such chemical intervention in the treatment of immunodeficiency syndromes. Young adult and aging (18-month-old) male rats were orchidectomized under ether anesthesia and 7 days later given s.c. implants of testosterone in silicone elastomer (SILASTIC) tubing. Some rats received testosterone together with a five-fold excess of the aromatase inhibitor 1,4,6-androstatriene-3,17-dione (ATD). One group of young intact rats received implants containing 25 mg ATD and group of 18-month-old intact rats received 125 mg ATD or 25 mg of another, more powerful aromatase inhibitor 4-hydroxyandrostenedione (4-OH). On the 28th day after implanting, rats were killed and the thymus, spleen, prostate gland and seminal vesicles removed for weighing and histology. In addition, estrogen receptors were measured in the thymus was enlarged after orchidectomy and greatly restored in aging rats. In aging rats, both aromatase inhibitors restored the thymus, which appeared normal histologically. In addition, ATD enlarged the thymus in young intact animals. ... It is therefore possible to restore the thymus in intact aging rats without recourse to surgical or chemical castration, and such a maneuver may possibly be of use to enhance an immune system weakened by aging or disease."
>>77247900
>>77247899>Potential repositioning of exemestane as a neuroprotective agent for Parkinson's diseasehttps://sci-hub.ru/https://pubmed.ncbi.nlm.nih.gov/28770670/The present study examined for the first time the effect of exemestane on the brain. Exemestane is highly lipophilic, and it distributes extensively into tissues, owing to its lipophilicity [59], suggesting that it can readily enter the brain. Our results demonstrated that exemestane activate the Nrf2 signalling pathway, induce gene expression of Nrf2-depensent enzyme genes, and suppress inflammatory responses. It also prevented microglial activation, dopaminergic neurodegeneration, and motor deficits in MPTP-treated mice. Therefore, exemestane repositioning for disease-modifying therapy for PD might be considered, in addition to its current usage in breast cancer.
>>77247906>Association Between Hormone-Modulating Breast Cancer Therapies and Incidence of Neurodegenerative Outcomes for Women With Breast Cancerhttps://jamanetwork.com/journals/jamanetworkopen/fullarticle/2763234In this cohort study of 57843 perimenopausal- to postmenopausal-aged women with breast cancer, exposure to hormone-modulating therapy (tamoxifen and aromatase inhibitors, especially exemestane) was associated with a significant decrease in the number of women who received a diagnosis of neurodegenerative disease, most specifically Alzheimer disease.For AD, HMTs (such as estrogen therapy) have been shown to be associated with the onset of AD,34 whereas estrogen therapy has been shown to be ineffective as a treatment for those with a diagnosis of AD.35-43 Similarly, the failure of trials evaluating the use of SERMs in the treatment of AD could be due to the fact that the intervention was started past the therapeutic window for HMTs.18,44-46 Here, we show the beneficial effects of exposure to HMT as a prophylactic treatment for the potential prevention of AD.
>>77247908
>>77247487>straight from the steppesCorrect, literally straight from the land where genesis of aryan race happened.
>>77247941>Admits being indianOH NONONONO
>>77247069>>77247135Why aren't you trying to cure your autism?
>>77247848What's the function of aromatase in male physiology?
>>77248447To produce estrogen from testosterone. Estrogen is needed partly in adult life, but mostly it's needed to produce some testosterone-dependent sexual dimorphism in puberty (as evident by aromatase-null males) - after that, not so much, It's similar to DHT in that regard. Even in puberty if you have too much conversion of testosterone to estrogen via aromatase you can develop gyno or low height.
>>77247052>Finasteride
>>77248536IM SO FULL FROM FINASTERIDE YUM
>>77248536
>>77248489Dude... no...>To produce estrogen from testosterone.This is how aromatase performs its role. The role of aromatase in male physiology is to ensure testosterone homeostasis. It does this in two ways. Testosterone in circulation inhibits testosterone production by inhibiting LH in the hypothalamus. Aromatization of testosterone in circulation is one of the mechanisms that lifts this inhibition, prompting the release of GnRH and LH by the hypothalamus, which are required to stimulate cells in the male testes responsible for spermatogensis and the majority of testosterone synthesis. Inhibition of aromatase impairs GnRH and LH release, and inhibits steady testosterone levels.Aromatase is also required for testosterone to exert its effects on male brains. Throughout adulthood, key areas in the male brain involved in memory (hippocampus), sexual behaviour (pre-optic area), pain tolerance (anterior cingulate cortex) and emotional regulation (DLPFC) are regulated exclusively by estrogen receptors. Testosterone is locally aromatized, which is the only manner in which testosterone can exert its effects on male behaviour by masculinizing the architecture of the male brain (throughout adulthood).The same mechanism is responsible for controlling the suppression of LH in the hypothalamus. LH is suppressed by testosterone itself, and testosterone which is locally aromatized. Estrogen in circulation, produced through aromatization, is blocked from interacting with estrogen receptors in the hypothalamus. So, these are two different, separate mechanisms of aromatization of testosterone.
>>77247908>>77248489>>77248594Additionally, you are referencing a study of aromatase inhibition in females to allude to its suggested beneficial effects in males, but this is a grave, grave mistake.The role of aromatase in the female body is DRASTICALLY different from that of males.Aromatase in the female body is responsible for the production of estrogen. All estrogen is fabricated from testosterone, including the female body. Until methods were discovered to synthesize testosterone in vitro in the 80s, testosterone was harvested from pregnant women in the 60s and 70s, since pregnancy drastically elevates sex steroid production, for which testosterone is the precursor. In the past, pregnancy tests worked through the detection of testosterone and epitestosterone in female urine. Aromatase functions as a homeostatic control mechanism in the male nervous system. Aromatase functions as the agent for sex steroid production in females. The problem with post-menopausal females and Alzheimer's is that there are multiple forms of estrogen. Estradiol is crucial in maintaining cognitive function in female brains, and estradiol levels decline significantly with menopause. The problem is not levels of estrogen itself, its reduced levels of estradiol compared to estrone. The point of aromatase inhibitors in females in the context of Alzheimer's is to reduce levels of estrone, increasing the ratio of estradiol to estrone (which will keep being produced in the same quantities). As you can imagine, since male brain physiology is regulated through local aromatization of testosterone, estrogen in circulation is excluded from interacting with estrogen receptors, and males don't have a physiology where the management of estradiol and estrone is present, aromatase inhibitors will not interact with the neurobiological characteristics of Alzheimer's the way they do in post-menopausal women.
>>77247908>>77248489>>77248594>>77248601In general, hormonal regulation as it relates to behaviour and brain function is remarkably different in males and females.Male sex steroid regulation works predominately through negative feedback. Testosterone blocks testosterone production. Activity in estrogen receptors in the brain reduces the amount of estrogen receptors. Male sex steroid regulation is characterized by the nervous system attempting to maintain steady levels of testosterone production and testosterone in circulation, reducing peaks and dips. Testosterone's effects on male behaviour in the brain are mediated through estrogen receptors, which acts a "safeguard" to ensure that excess testosterone does not lead to changes in male physiology that further affect testosterone production. Testosterone is still required to make them happen, but the degree to which it can bring about changes is controlled by aromatization. Inhibition of aromatase not only inhibits aromatase of testosterone in the circulation (a crucial process, which in and of itself is responsible for initiating production of new testosterone), inhibition of aromatase also prevents testosterone from making male brains "more male".Female sex steroid regulation works predominately through positive feedback. Estrogens in circulation don't inhibit LH, they stimulate LH. This is how ovulation happens, because sex steroids in females stimulate the production of more sex steroids, until the system is overloaded (which initiates the process of menstruation). As you can imagine, aromatase is not a stopgap mechanism in female physiology used to maintain stable levels of sex steroids. (additionally, this is why trannies can never ovulate, because the anatomy of the female hypothalamus is different from the anatomy of the male hypothalamus, since the male hypothalamus is keyed towards sex steroids providing negative feedback, while ovulation depends on positive feedback mechanisms.
>>77247908>>77248489>>77248594>>77248601>>77248614You reference a study on the use of aromatase inhibitors in the management of Alzheimer's in post-menopausal women, in support of using aromatase inhibitors for male cognitive performance and well-being. The fact that you did reveals some serious misunderstandings about male physiology and female physiology. Long-term ingestion of various substances you've listed will lead to serious harm, if these substances are ingested based on your lack of understanding of human endocrinology, biochemistry and molecular microbiology.You don't know what you're doing. You're harming yourself. You're harming others by trying to push your ideas on them, based on serious shortcomings in your knowledge of basic human biology.All the time you've invested in looking at these articles, quoting them, you could easily have invested in actually reading a book on this subject. The same time. The same effort.Please, instead of posting your shit here, spend that same amount of time and effort just opening the book I'm linking, and start reading it. It's a really good book. If you're able to keep up with it, you can learn things you can actually use in the contexts you describe in these threads. If you're not able to keep up with the material, that should tell you how little you adequately understand the contents of the studies you list, and the workings of the substances you are listing.https://doku.pub/documents/anintroductiontobehavioralendocrinolpdf-nl31p8gdgvq1Hopefully you appreciate the effort I'm putting into writing this. I'm genuinely concerned for you or anyone who takes up your suggestions.
>>77248626What's your take on Telmisartan? I'm taking it myself to prevent cardiac remodeling from Nandrolone.
>>77248594>The role of aromatase in male physiologyHonestly, i'm not of the mindset to think of the hormones as having different "roles" in the body. It's all chaotic chemical reactions, which can sometimes produce unexpected and counterintuitive results. Speaking of them as having "roles" is either just a simplistic way of communication or creationist retardation. >Inhibition of aromatase impairs GnRH and LH release, and inhibits steady testosterone levels.It's not, in fact the opposite is true. Look into case studies of human males with genetic aromatase deficiency. >Aromatase deficiency in male and female siblings caused by a novel mutation and the physiological role of estrogens https://sci-hub.ru/10.1210/jc.80.12.3689>The role of estrogens on the sex steroid-gonadotropin feedback mechanism is clarified by the observations in patients with P450arom deficiency. The elevated levels of androgens in our two patients as well as in the female described by Conte et al. (4) failed to adequately suppress gonadotropins after the age of puberty. ... The elevated (but not castrate) concentrations of plasma FSH and LH in the brother despite the strikingly high circulating T and dihydrotestosterone levels supports an important feedback role of estrogen, either of peripheral origin or synthesized locally from Cl9 steroid precursors or from both sources as well as T, in the regulation of FSH and LH in the male>The abnormally high (-2.5 times) plasma concentration of T is probably a consequence of the high plasma LH concentrations that induced either functional hyperleydigism or an increase in Leydig cell mass in the virtual absence of local E2 synthesis and circulating E2
>>77248594>>77249124>But it's just 1 mutantFine, let's see the studies measuring effect of aromatase inhibition on GnRH, LH and testosterone in normally developed men.>Pharmacokinetics and Dose Finding of a Potent Aromatase Inhibitor, Aromasin (Exemestane), in Young Maleshttps://www.researchgate.net/publication/8963583_Pharmacokinetics_and_Dose_Finding_of_a_Potent_Aromatase_Inhibitor_Aromasin_Exemestane_in_Young_MalesSuppression of estrogen, via estrogen receptor or aromataseblockade, is being investigated in the treatment of differentconditions. Exemestane (Aromasin) is a potent and selectiveirreversible aromatase inhibitor. To characterize its suppres-sion of estrogen and its pharmacokinetic (PK) properties inmales, healthy eugonadal subjects (14–26 yr of age) were re-cruited. In a cross-over study, 12 were randomly assigned to25 and 50 mg exemestane daily, orally, for 10 d with a 14-dwashout period. Blood was withdrawn before and 24 h after the last dose of each treatment period. A PK study was per-formed (n ⴝ 10) using a 25-mg dose. Exemestane suppressed plasma estradiol comparably with either dose [25 mg, 38% (P <0.002); 50 mg, 32% (P < 0.008)], with a reciprocal increase in testosterone concentrations (60% and 56%; P < 0.003 for both).Plasma lipids and IGF-I concentrations were unaffected by treatment. The PK properties of the 25-mg dose showed the highest exemestane concentrations 1 h after administration, indicating rapid absorption. The terminal half-life was 8.9 h. Maximal estradiol suppression of 62 ⴞ 14% was observed at12 h. The drug was well tolerated. In conclusion, exemestaneis a potent aromatase inhibitor in men and an alternative to the choice of available inhibitors.
>>77248594>>77249124>>77249138>The effect of testosterone aromatization on high-density lipoprotein cholesterol level and postheparin lipolytic activity.https://pismin.com/10.1016/0026-0495(93)90101-s
>>77249124>>77249138>>77249152Meta-analysis of aromatase inhibitors usage in men. >Aromatase inhibitors in men: effects andtherapeutic optionshttps://www.wellesu.com/10.1186/1477-7827-9-93
>>77248601>The point of aromatase inhibitors in females in the context of Alzheimer's is to reduce levels of estrone, increasing the ratio of estradiol to estrone (which will keep being produced in the same quantities).Exemestane, aromatase inhibitor that showed best results in preventing alzheimer's disease and related dementias in the study i cited, often reduces both estradiol and estrone to the same level, and in some studies reduces estradiol even more than estrone. >New aromatase inhibitors for the treatment of advanced breast cancer in postmenopausal womenhttps://www.sciencedirect.com/science/article/abs/pii/S0300297799000303>In healthy postmenopausal women and breast cancer patients oral doses of 0.5–800 mg o.d. suppressed plasma estradiol with 61–85%, estrone with 65–94% >Effects of exemestane and letrozole therapy on plasma concentrations of estrogens in a randomized trial of postmenopausal women with breast cancerhttps://pmc.ncbi.nlm.nih.gov/articles/PMC5429096/#S13Month-3 concentrations of E2, E1, and E1S fell below assay LLOQ in 87.9%, 85.1%, and 36.3% of patients, respectively (Table 1). The frequency of E2 suppression below LLOQ was not statistically significantly different between AIs (exemestane: 89.0%, letrozole: 86.9%, p=0.51); however, significant differences were observed for both E1 and E1S. E1 concentrations were reduced below the assay LLOQ in 90.1% of patients taking letrozole, compared to 80.1% of patients taking exemestane (p=0.005). Similarly, although 54.9% of patients taking letrozole had month-3 E1S concentrations below the assay LLOQ, this reduction was only observed in 17.4% of patients taking exemestane (p=4.34e-15).
>>77248626>You don't know what you're doing. You're harming yourself. You're harming others by trying to push your ideas on them, based on serious shortcomings in your knowledge of basic human biology.Considering you can't even double-check your own made-up assertions, i diagnose you with severe case of mental retardation, and conclude you must receive help and guidance from qualified medical professional, including prohibition of internet usage to prevent further harm to yourself or others. Perish, mortal, for the art of alchemy will forever be outside your comprehension.
>>77249236Seriously m8, did you get an official autism diagnosis? It's very obvious that you have it (I do too) and you need to work on yourself.
>>77249248>did you get an official autism diagnosis?I'm definitely not neurotypical, but it's not autism since i don't have problems at socializing when i want to or adapting to new environments - probably psychopathy, narcissism, megalomania and high trait dopamine always making me excited to go down the rabbit holes of things i'm interested in.
>>77248601>>77249207As to actually plausible explanations of beneficial effects of aromatase inhibitors in prevention of neurodegenerative diseases...>Neuroprotection from raised endogenous androgens>Neuroprotection from androgenic metabolites of aromatase inhibitors (such as 17B-hydroexemestane in case of exemestane)>Slowing of thymic involution>Decreasing of toxic metabolites of estrogen>Off-target effects (such as Nrf2 upregulation by exemestane)
>>77247052Troon general
>>77247075>with high cholesterol it doesn't matter how many other things you take, you'll still die early.There's many more healthy ways to diminish LDL and damage done by it besides statins, including drugs of sartan class, ezetimibe that prevents alzheimer's disease by improving neural protein dynamics as off-target effect, anti-inflammatory drugs (such as aspirin that also raises free thyroid hormones), and direct thyroid-mimetics such as sobetirome. >Best Sobetirome (GC-1) Protocols (Lipid-Lowering Anti-Catabolic T3 Thyroid Alternative)https://www.youtube.com/watch?v=QaWidRqxGOM
>>77247052>Magnesium>no borox or boron
>>77249834I know, i just can't source boron at a reasonable price and with certainty that it's a real product.
>>77249884just take borax its desert salt
>>77249929Are you sure it's safe and effective, anon-kun...>Soldering of copper, brass, cast iron, steel>Removal of stains and mold>Universal disinfectantThey all sell technical-grade borax. There may be impurities left from technological production process.
I take>creatine>arginine>vitamin D>olive oil>black tea>nutmeg>cumin>turmeric
>>77249951Normiest stack ever.
>>77249939The Borax Conspiracy How the Arthritis Cure has been Stopped - Walter Last
>>77249951>turmericIndian scam.
>>77247052Just made new anabolic cacao jelly full of (-)-epicatechin. Ingredients: 50g bovine collagen powder70g trehalose75g pure cacao powder>Cacao flavonoid (-)-epicatechin inhibits myostatin and strengthens muscleshttps://www.ergo-log.com/cacao-flavonoid-epicatechin-inhibits-myostatin-strengthens-muscles.htmlTreatment for 7 days with (-)-epicatechin yielded a bilateral increase in hand strength of 7%, which was accompanied by a significant increase (49.2%) in the ratio of plasma follistatin/myostatin levels"
I'm the guy from the last thread who asked about bigger balls. I don't have access to HCG or any of the other stuff that would probably make it happen faster, but I did start taking iron (27mg), D3 (5000 IU), and zinc (50mg). It's not as much as the Russian guy, but it's what I could get off the shelf.I'll let y'all know if anything comes of it, but so far I think the iron is just giving me fatigue. I don't have any other symptoms of iron toxicity so it may be some other energy problem.
>on a literal kitchen sink worth of pharmaceuticals but refuses to pin because "muh hpta"You can get all the benefits of your drug stack by just doing cardio, lifting, and having sub 15% bodyfat if you're not gonna do anabolics..kek
>>77249496Very based
>>77250741>You can get all the benefits of your drug stack by just doing cardio, lifting, and having sub 15% bodyfatNo, you can't. From my stack, only telmisartan and bromocriptine have slight exercise-mimetic properties - everything else acts trough different pathways.
>>77250793>ExemestaneLower the bodyfat and you'll have less aromatase expression, no AI needed>aspirinLower bodyfat and proper diet/hydration lowers hematocrit and thus blood thickness>montelukastUpping your VO2max through cardio increases vasodilation and lung efficiency, and effectively reduces systemic inflammation>bromocriptineThere is no reason for you to be taking a dopamine agonist. Have fun trying to get off of that when you inevitably will have to in the future>finasterideIf you're trying not to be bald then this is literally the only valid medication on hereYou are trying to pill your way out of putting the work into diet, training, and consistency. Pathetic.
>>77250842You have a trait very indicative of a low intelligence, namely - no sense of magnitude and nuance. Retards like you usually always seek natural solutions to their problems (like using saw palmetto instead of finasteride because "it inhibits 5-alpha reductase too bro!"), only to fail because they have 1/10 the power of drugs.
>>77250744Agree. Appreciating trans-women is /fit/ culture.
>>77247487you wish you were slavic, brownoid
>>77250744adorable snek drinkk
