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what no one is talking about right now is that ANDV is segmental virus, its genome is broken into multiple segments, each responsible for different attributes. Segmental viruses in a family (like influenza or in this case hantaviruses) do the viral equivalent of sexual reproduction when they coinfect the same host, during Virion assembly the segments from viruses within a family get shuffled. Unfortunately for us the pathogenic behavior and h2h transmission potential of the Andes virus is located on the M segment, the most easily interchanged segment (S and L segments are codependent and must come from the same parent) and the most durable, environmentally persistent Hantavirus is the most common one in Europe (Puumala virus) present in the most common mammal in Europe, the vole along with secondary reservoirs in mice in Europe, its persistence is a result of its s segment, it is also the most accepting of reassorted m segments of any hantavirus, even accepting reassortment with prospect hill virus despite it being a new world hantavirus (the same as ANDV) and PUUV being a old world hantavirus, with no real loss of offspring virion viability. if ANDV spreads in europe its a matter of WHEN not IF, ANDV will reassort with PUUV.

when that happens the intrinsic background mutation rate of resultant reassortant hybrid will 10x, it will posses all the; human to human transmission ability, HPS related lethality of ANDV but will also increase environmental lifespan from 3 days to 15--18 days resulting in not just a increased exposure window but a 500% increase in peak virion exposure concentration because the shed viral particles can accumulate over that full 15 day period.

ANDV has not met PUUV in the past and now they're about too.